Carcinogenesis.178 In these settings, IL-1 and MyD88 (but not IL-1) activation trigger the loss of keratinocyte differentiation and promote invasiveness. Similar results have previously been found in melanoma patients, where IL-1 induced the MyD88dependent activation of the NF-B and MAPK pathways as well as an increase in ROS production, thereby promoting tumor progression.179 Furthermore, in panc
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